Smad6 and Smad7 are co-regulated with hepcidin in mouse models of iron overload

Erstveröffentlichung
2013Autoren
Spasic, Maja Vujic
Sparla, Richard
Mleczko-Sanecka, Katarzyna
Migas, Mary C.
Breitkopf-Heinlein, Katja
Wissenschaftlicher Artikel
Erschienen in
Biochimica et biophysica acta : Molecular basis of disease ; 1832 (2013), 1. - S. 76-84. - ISSN 0925-4439. - eISSN 1879-260X
Link zur Veröffentlichung
https://dx.doi.org/10.1016/j.bbadis.2012.08.013Schlagwörter
[Freie Schlagwörter]: Smad6 | Smad7 | Hepcidin | Hemochromatosis | Iron | Bmp/Smad | TRANSFERRIN RECEPTOR 2 | HEREDITARY HEMOCHROMATOSIS | TARGETED DISRUPTION | SIGNAL-TRANSDUCTION | DUODENAL HFE | MICE LEADS | EXPRESSION | METABOLISM | GENE | HOMEOSTASIS[DDC Sachgruppe]: DDC 570 / Life sciences