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Hepcidin knockout mice spontaneously develop chronic pancreatitis owing to cytoplasmic iron overload in acinar cells

Erstveröffentlichung
2017
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Wissenschaftlicher Artikel


Authors
Lunova, Mariia
Schwarz, Peggy
Nuraldeen, Renwar
Levada, Kateryna
Kuscuoglu, Deniz
et al.
Faculties
Fakultät für Naturwissenschaften
Institutions
UKU. Klinik für Innere Medizin I
Institut für Molekulare Endokrinologie der Tiere
Published in
Journal of Pathology ; 241 (2017), 1. - S. 104-114. - ISSN 0022-3417. - eISSN 1096-9896
Link to publication
https://dx.doi.org/10.1002/path.4822
Keywords
chronic pancreatitis; iron overload; minihepcidin; hereditary hemochromatosis; transfused children; liver fibrosis; mouse models; metabolism; inflammation; transferrin; homeostasis; gene; accumulation
Dewey Decimal Group
DDC 610 / Medicine & health

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