Hepcidin knockout mice spontaneously develop chronic pancreatitis owing to cytoplasmic iron overload in acinar cells

Lunova, Mariia; Schwarz, Peggy; Nuraldeen, Renwar; Levada, Kateryna; Kuscuoglu, Deniz; Stuetzle, Michael; Vujic Spasic, Maja; Haybaeck, Johannes; Ruchala, Piotr; Jirsa, Milan; Deschemin, Jean-Christophe; Vaulont, Sophie; Trautwein, Christian; Strnad, Pavel

Erstveröffentlichung

2017

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Wissenschaftlicher Artikel

Authors

Lunova, Mariia

Schwarz, Peggy

Nuraldeen, Renwar

Levada, Kateryna

Kuscuoglu, Deniz

et al.

Faculties

Fakultät für Naturwissenschaften

Institutions

UKU. Klinik für Innere Medizin I
Institut für Molekulare Endokrinologie der Tiere

Published in

Journal of Pathology ; 241 (2017), 1. - S. 104-114. - ISSN 0022-3417. - eISSN 1096-9896

Link to publication

https://dx.doi.org/10.1002/path.4822

Funding information

Interdisciplinary Centre for Clinical Research (IZKF) within the faculty of Medicine at the RWTH Aachen University
Deutsche Forschungsgemeinschaft (DFG) [SFB TRR57]
Ministry of Health, Czech Republic - conceptual development of research organization
Else Kroner Exzellenzstipendium

Keywords

chronic pancreatitis; iron overload; minihepcidin; hereditary hemochromatosis; transfused children; liver fibrosis; mouse models; metabolism; inflammation; transferrin; homeostasis; gene; accumulation

Dewey Decimal Group

DDC 610 / Medicine & health

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