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MEIS2 is a novel oncogenic partner in AML1-ETO positive AML
Universität Ulm (2016)
Forschungsdaten
GSE81328: MEIS2 is a novel oncogenic partner in AML1-ETO positive AML [RNA-Seq human]
Mulaw, Medhanie Assmelash (2016)
Forschungsdaten
Domain mapping of full-length huntingtin by limited proteolysis with chymotrypsin (2016/03/15)
Harding, Rachel; Huang, Bin; Kochanek, Stefan; Toledo-Sherman, Leticia; Arrowsmith, Cheryl; Edwards, Aled (2016)
Forschungsdaten
Analysis of huntingtin methylations by mass spectrometry (2016/08/30)
Harding, Rachel; Ackloo, Suzanne; Taylor, Paul; Kochanek, Stefan; Toledo-Sherman, Leticia; Arrowsmith, Cheryl; Edwards, Aled (2016)
Forschungsdaten
Domain mapping of full-length huntingtin by limited proteolysis with chymotrypsin (2016/03/16)
Harding, Rachel; Huang, Bin; Kochanek, Stefan; Toledo-Sherman, Leticia; Arrowsmith, Cheryl; Edwards, Aled (2016)
Forschungsdaten
GSE77919: ALS-causing mutations differentially affect PGC-1alpha expression and function in the brain vs. peripheral tissues
Bayer, Hanna; Holzmann, Karlheinz; Witting, Anke (2016)
Forschungsdaten
GSE81329: MEIS2 is a novel oncogenic partner in AML1-ETO positive AML [RNA-Seq mouse]
Mulaw, Medhanie Assmelash (2016)
Forschungsdaten
GSE81321: MEIS2 is a novel oncogenic partner in AML1-ETO positive AML [ChIP-Seq]
Mulaw, Medhanie Assmelash (2016)
Forschungsdaten
GSE81174: MEIS2 is a novel oncogenic partner in AML1-ETO positive AML [Affymetrix]
Mulaw, Medhanie Assmelash (2016)
Forschungsdaten
GSM2144148: KL_C (AE_Meis2)
Mulaw, Medhanie Assmelash (2016)
Forschungsdaten