OPARU is the OPen Access Repository of Ulm University and Ulm University of Applied Sciences.
Members and affiliates of Ulm University and Ulm University of Applied Sciences can publish via OPARU and thereby make their publications accessible worldwide free of charge. In addition to traditional publication formats, research data can also be published on OPARU. Publications can also be indexed as "metadata only" entries. This means that publication lists can be completely integrated in OPARU.
The bibliography of Ulm University and the bibliography of Ulm University of Applied Sciences are integrated in OPARU and include all publications that originated at the respective institution. Both bibliographies are continuously maintained by the kiz and the THU, respectively.
Via OPARU you can comply with the recommendation of the Open Access Resolution of Ulm University and the Open Access Policy of the THU to self-archive your publications on the institutional repository.
Learn more about OPARU (instructions, features...)
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Cryo-EM demonstrates the in vitro proliferation of an ex vivo amyloid fibril morphology by seeding (2022)Wissenschaftlicher Artikel
Gc at its boundaries: A cross-national investigation of declarative knowledge (2023)Wissenschaftlicher Artikel
Conservation measures or hotspots of disease transmission? Agri-environment schemes can reduce disease prevalence in pollinator communities (2023)Wissenschaftlicher Artikel
Perspective on quantum bubbles in microgravity (2023)Wissenschaftlicher Artikel
The potential of zero charge of a metal electrode and the surface potential of water from simulations (2023)Wissenschaftlicher Artikel
Efimovian three-body potential from broad to narrow Feshbach resonances (2023)Wissenschaftlicher Artikel
Loss of SUV420H2-Dependent Chromatin Compaction Drives Right-Sided Colon Cancer Progression (2023)Wissenschaftlicher Artikel
What we don't need to prove but need to do in multidisciplinary treatment and care in Huntington's disease: a position paper (2023)Wissenschaftlicher Artikel
Npm1 haploinsufficiency in collaboration with MEIS1 is sufficient to induce AML in mice (2023)Wissenschaftlicher Artikel
JAML immunotherapy targets recently activated tumor CD8(+) T cells (2023)Wissenschaftlicher Artikel